toyoshima hideo

FacultyComprehensiveMedicine1
Job titleAssociate ProfessorWeb site

Profile

Research Areas

  • Biological science / Cell biology
  • Clinical internal medicine / Metabolomics

Career

  • 1990
    -
    1996
     Assistant Professor, Faculty of Medicine,University of Tokyo

Academic Background

  • -
    1985
    Faculty of Medicine, The University of Tokyo (japan)

Research Activities

Papers(other)

  • p57Kip2 is degraded through the proteasome in osteoblasts stimulated to proliferation by transforming growth factor β1.
    Journal of Biological Chemistry 274(18) 12197-12200 1999
  • Inctivation of p27Kip1 by the viral E1A oncoprotein in TGF β-treated cells.
    Nature 380(6571) 262-265 1996
  • p27, a novelinhibitor of G1 cyclin-Cdk protein kinase activity, is related to P21.
    Cell 78(1) 67-74 1994
  • Role of cyclin A and p27 in anti-IgM-induced G1 growth arrest of murine B-cell lymphomas.
    Molecular Biology of the Cell 7 553-564 1996
  • Cyclin-dependent Kinases are inactivated by a combination of p21 and Thr-14/Tyr-15 phosphorylation after UV-induced DNA damage.
    Journal of Biological Chemistry 271(22) 13283-13291 1996
  • Cyclin-dependent kinases are inactivated by a combination of p21 and Thr-14/Tyr-15 phosphorylation after UV-induced DNA damage.
    Journal of Biological Chemistry 271(22) 13283-13291 1996
  • Redistribution of the CDK inhibitor p27 between different cyclin・CDK complexes in the mouse fibroblast cell cycle and in cells arrested with lovastatin or ultraviolet irradiation.
    Molecular Biology of the Cell 6 1197-1213 1995
  • Differently spliced cDNAs of human ltk receptor tyrosine kinase predict receptor proteins with and without tyrosine kinase domain and a soluble receptor protein.
    Proceedings of the National Academy of Sciences of the United States of America 90(12) 5404-5408 1993
  • Secondary structure of the human T-cell leukemia virus type 1 rex-responsive element is essential for rex regulation of RNA processing and trnsport of unspliced RNAs.
    Journal of Virology 64 2825-2832 1990
  • Smad-mediated Transcription Is Required for Transforming Growth Factor-β1-induced p57Kip2 Proteolysis in Osteoblastic Cells
    The Journal of Biological Chemistry 276(14) 10700-10705 2001
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Papers(other)close
  • p57Kip2 is degraded through the proteasome in osteoblasts stimulated to proliferation by transforming growth factor β1.
    Journal of Biological Chemistry 274(18) 12197-12200 1999
  • Inctivation of p27Kip1 by the viral E1A oncoprotein in TGF β-treated cells.
    Nature 380(6571) 262-265 1996
  • p27, a novelinhibitor of G1 cyclin-Cdk protein kinase activity, is related to P21.
    Cell 78(1) 67-74 1994
  • Role of cyclin A and p27 in anti-IgM-induced G1 growth arrest of murine B-cell lymphomas.
    Molecular Biology of the Cell 7 553-564 1996
  • Cyclin-dependent Kinases are inactivated by a combination of p21 and Thr-14/Tyr-15 phosphorylation after UV-induced DNA damage.
    Journal of Biological Chemistry 271(22) 13283-13291 1996
  • Cyclin-dependent kinases are inactivated by a combination of p21 and Thr-14/Tyr-15 phosphorylation after UV-induced DNA damage.
    Journal of Biological Chemistry 271(22) 13283-13291 1996
  • Redistribution of the CDK inhibitor p27 between different cyclin・CDK complexes in the mouse fibroblast cell cycle and in cells arrested with lovastatin or ultraviolet irradiation.
    Molecular Biology of the Cell 6 1197-1213 1995
  • Differently spliced cDNAs of human ltk receptor tyrosine kinase predict receptor proteins with and without tyrosine kinase domain and a soluble receptor protein.
    Proceedings of the National Academy of Sciences of the United States of America 90(12) 5404-5408 1993
  • Secondary structure of the human T-cell leukemia virus type 1 rex-responsive element is essential for rex regulation of RNA processing and trnsport of unspliced RNAs.
    Journal of Virology 64 2825-2832 1990
  • Smad-mediated Transcription Is Required for Transforming Growth Factor-β1-induced p57Kip2 Proteolysis in Osteoblastic Cells
    The Journal of Biological Chemistry 276(14) 10700-10705 2001

Outside Fund Adoption

  • Cell Differentiation and Cell Cycle in Atherosclerosis
    :The Other Research Programs
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Outside Fund Adoptionclose
  • Cell Differentiation and Cell Cycle in Atherosclerosis
    :The Other Research Programs


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